Search results for "Allyl Mercaptan"

showing 2 items of 2 documents

Hepatic metabolism of diallyl disulfide in rat and man

2003

International audience; 1. The metabolism of diallyl disulphide was investigated in vitro with rat and human liver cell subfractions and ex vivo with an isolated perfused rat liver. 2. Diallyl disulphide was oxidized to diallylthiosulphinate by rat liver microsomes with an apparent K-m = 0.86 +/- 0.1 mM and an apparent V-max = 0.47 +/- 0.12 nmol min(-1) mg(-1) protein (mean +/- SE). Both cytochrome P450 (CYP) and flavin-containing monooxygenases were involved, with CYP2B1/2 and CYP2E1 being the most active CYP enzymes. 3. In rat and man, microsomal oxidation of allylmethyl sulphide to allylmethyl sulphoxide and allylmethyl sulphone also occurred, although at a low rate. Diallyl disulphide w…

MaleLIVERHealth Toxicology and MutagenesisToxicologyBiochemistryGARLICchemistry.chemical_compoundDisulfides0303 health sciencesbiologyDADS030302 biochemistry & molecular biologyCytochrome P-450 CYP2E1General MedicineCYP2E1Middle Agedfoie3. Good healthEnzymesAllyl CompoundsPerfusionBiochemistryArea Under CurveMicrosomes LiverFemaleAryl Hydrocarbon HydroxylasesOxidation-Reductionailcomposé soufrexénobiotique[SDV.BID]Life Sciences [q-bio]/BiodiversityIn Vitro Techniques03 medical and health sciencesAnimalsHumansmétabolisme030304 developmental biologyAgedPharmacologySulfur CompoundsEX VIVOCytochrome P450SULFUR COMPOUNDMetabolismGlutathioneMonooxygenaseRatschemistryDADS;EX VIVO;SULFUR COMPOUND;XENOBIOTIC METABOLISM;GARLIC;LIVER;RATCytochrome P-450 CYP2B1Steroid HydroxylasesMicrosomebiology.proteinRATAllyl MercaptanXENOBIOTIC METABOLISMDrug metabolism
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In vivo antigenotoxic effects of dietary allyl sulfides in the rat

1997

The effects of dietary administration of diallyl sulfide (DAS), diallyl disulfide (DADS) and allyl mercaptan (AM) on the genotoxicity of different chemicals were studied in two experimental systems: (i) measurement of hepatic DNA single-strand breaks induced in rats by aflatoxin B1 (AFB1), N-nitrosodimethylamine (NDMA) or methylnitrosourea (MNU); (ii) mutagenicity of AFB1 or NDMA on Salmonella typhimurium TA100 using hepatic S9 from rats fed allyl sulfides as the activation system. All compounds strongly reduced hepatic DNA breaks induced by AFB1 and NDMA but did not modify the genotoxicity of MNU. In the Ames test, the mutagenicity of NDMA was strongly inhibited by hepatic S9 from rats fed…

MaleSalmonella typhimuriumCancer ResearchAflatoxin B1[SDV]Life Sciences [q-bio]Allyl compoundMutagenSulfidesmedicine.disease_cause030226 pharmacology & pharmacyDimethylnitrosamineAmes test03 medical and health scienceschemistry.chemical_compound0302 clinical medicineN-NitrosodimethylaminemedicineAnimalsAnticarcinogenic AgentsDisulfidesComputingMilieux_MISCELLANEOUSMutagenicity TestsDiallyl disulfidefood and beveragesAntimutagenic AgentsMethylnitrosoureaRats3. Good healthAllyl Compounds[SDV] Life Sciences [q-bio]LiverOncologychemistryBiochemistry030220 oncology & carcinogenesisRATAllyl MercaptanCARCINOGENESEAllyl SulfideGenotoxicityDNA DamageMutagensCancer Letters
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